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mouse anti human vimentin primary antibodies  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc mouse anti human vimentin primary antibodies
    <t>PGI</t> expression was increased in hyperfibrotic regions in keloid tissues. (A) Immunohistochemical staining for PGI in normal skin and keloids (original magnification 40). Right panels are high-power views of the tissues (original magnification 200). The black rectangles in the left panels indicate the areas enlarged at high magnification, respectively. The PGI intensity is shown (B). (C) Sections of keloid tissues and normal skin were deparaffinized and further incubated with antibodies against PGI and <t>vimentin.</t> The expression of PGI (green) and vimentin (red) was localized in keloid tissue and normal skin (original magnification 100). Right panels are high-power views of tissues, identifying colocalization of PGI (green) and vimentin (red) in keloid and normal skin (original magnification 200). The white rectangles in the left panels show the enlarged areas at high magnification, respectively. The number of PGI-positive, vimentin-expressing cells was quantified from five randomly selected high-power fields. The percentage of these cells normalized to total cells (DAPI-stained cells) in the hypercellular areas is shown (D). Data are presented as the mean ± SD. (** p < 0.01) from representative data of 3 independent experiments.
    Mouse Anti Human Vimentin Primary Antibodies, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 98/100, based on 4551 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+anti+human+vimentin+primary+antibodies/Vimentin+XP+Rabbit+mAb/pmc12826743-90-13-19
    Average 98 stars, based on 4551 article reviews
    mouse anti human vimentin primary antibodies - by Bioz Stars, 2026-10
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    Images

    1) Product Images from "Phosphoglucose isomerase directs the inflammatory response, calcium influx and fibroblast migration in keloids"

    Article Title: Phosphoglucose isomerase directs the inflammatory response, calcium influx and fibroblast migration in keloids

    Journal: Future Science OA

    doi: 10.1080/20565623.2026.2615968

    PGI expression was increased in hyperfibrotic regions in keloid tissues. (A) Immunohistochemical staining for PGI in normal skin and keloids (original magnification 40). Right panels are high-power views of the tissues (original magnification 200). The black rectangles in the left panels indicate the areas enlarged at high magnification, respectively. The PGI intensity is shown (B). (C) Sections of keloid tissues and normal skin were deparaffinized and further incubated with antibodies against PGI and vimentin. The expression of PGI (green) and vimentin (red) was localized in keloid tissue and normal skin (original magnification 100). Right panels are high-power views of tissues, identifying colocalization of PGI (green) and vimentin (red) in keloid and normal skin (original magnification 200). The white rectangles in the left panels show the enlarged areas at high magnification, respectively. The number of PGI-positive, vimentin-expressing cells was quantified from five randomly selected high-power fields. The percentage of these cells normalized to total cells (DAPI-stained cells) in the hypercellular areas is shown (D). Data are presented as the mean ± SD. (** p < 0.01) from representative data of 3 independent experiments.
    Figure Legend Snippet: PGI expression was increased in hyperfibrotic regions in keloid tissues. (A) Immunohistochemical staining for PGI in normal skin and keloids (original magnification 40). Right panels are high-power views of the tissues (original magnification 200). The black rectangles in the left panels indicate the areas enlarged at high magnification, respectively. The PGI intensity is shown (B). (C) Sections of keloid tissues and normal skin were deparaffinized and further incubated with antibodies against PGI and vimentin. The expression of PGI (green) and vimentin (red) was localized in keloid tissue and normal skin (original magnification 100). Right panels are high-power views of tissues, identifying colocalization of PGI (green) and vimentin (red) in keloid and normal skin (original magnification 200). The white rectangles in the left panels show the enlarged areas at high magnification, respectively. The number of PGI-positive, vimentin-expressing cells was quantified from five randomly selected high-power fields. The percentage of these cells normalized to total cells (DAPI-stained cells) in the hypercellular areas is shown (D). Data are presented as the mean ± SD. (** p < 0.01) from representative data of 3 independent experiments.

    Techniques Used: Expressing, Immunohistochemical staining, Staining, Incubation

    Related Articles

    Incubation:

    Article Title: Phosphoglucose isomerase directs the inflammatory response, calcium influx and fibroblast migration in keloids
    Article Snippet: Excess proteins were blocked with Protein Block (RE7102, Leica). .. Sections were applied with 1:100-diluted rabbit anti-human PGI (#57893, Cell Signaling) and 1:500-diluted mouse anti-human vimentin primary antibodies (#5741, Cell Signaling) at 4 °C overnight followed by washing and incubated with secondary antibodies conjugated with 1:500-diluted goat anti-mouse Alexa Fluor TM 488 (A11001, Invitrogen) and goat anti-rabbit Alexa Fluor TM 568 (A11011, Invitrogen). .. After incubating with 1:1000-diluted DAPI staining (62248, Thermo Fisher) and Novolink Polymer. sections were counterstained with hematoxylin, dehydrated, and mounted with mounting medium.



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    <t>PGI</t> expression was increased in hyperfibrotic regions in keloid tissues. (A) Immunohistochemical staining for PGI in normal skin and keloids (original magnification 40). Right panels are high-power views of the tissues (original magnification 200). The black rectangles in the left panels indicate the areas enlarged at high magnification, respectively. The PGI intensity is shown (B). (C) Sections of keloid tissues and normal skin were deparaffinized and further incubated with antibodies against PGI and <t>vimentin.</t> The expression of PGI (green) and vimentin (red) was localized in keloid tissue and normal skin (original magnification 100). Right panels are high-power views of tissues, identifying colocalization of PGI (green) and vimentin (red) in keloid and normal skin (original magnification 200). The white rectangles in the left panels show the enlarged areas at high magnification, respectively. The number of PGI-positive, vimentin-expressing cells was quantified from five randomly selected high-power fields. The percentage of these cells normalized to total cells (DAPI-stained cells) in the hypercellular areas is shown (D). Data are presented as the mean ± SD. (** p < 0.01) from representative data of 3 independent experiments.
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    Image Search Results


    PGI expression was increased in hyperfibrotic regions in keloid tissues. (A) Immunohistochemical staining for PGI in normal skin and keloids (original magnification 40). Right panels are high-power views of the tissues (original magnification 200). The black rectangles in the left panels indicate the areas enlarged at high magnification, respectively. The PGI intensity is shown (B). (C) Sections of keloid tissues and normal skin were deparaffinized and further incubated with antibodies against PGI and vimentin. The expression of PGI (green) and vimentin (red) was localized in keloid tissue and normal skin (original magnification 100). Right panels are high-power views of tissues, identifying colocalization of PGI (green) and vimentin (red) in keloid and normal skin (original magnification 200). The white rectangles in the left panels show the enlarged areas at high magnification, respectively. The number of PGI-positive, vimentin-expressing cells was quantified from five randomly selected high-power fields. The percentage of these cells normalized to total cells (DAPI-stained cells) in the hypercellular areas is shown (D). Data are presented as the mean ± SD. (** p < 0.01) from representative data of 3 independent experiments.

    Journal: Future Science OA

    Article Title: Phosphoglucose isomerase directs the inflammatory response, calcium influx and fibroblast migration in keloids

    doi: 10.1080/20565623.2026.2615968

    Figure Lengend Snippet: PGI expression was increased in hyperfibrotic regions in keloid tissues. (A) Immunohistochemical staining for PGI in normal skin and keloids (original magnification 40). Right panels are high-power views of the tissues (original magnification 200). The black rectangles in the left panels indicate the areas enlarged at high magnification, respectively. The PGI intensity is shown (B). (C) Sections of keloid tissues and normal skin were deparaffinized and further incubated with antibodies against PGI and vimentin. The expression of PGI (green) and vimentin (red) was localized in keloid tissue and normal skin (original magnification 100). Right panels are high-power views of tissues, identifying colocalization of PGI (green) and vimentin (red) in keloid and normal skin (original magnification 200). The white rectangles in the left panels show the enlarged areas at high magnification, respectively. The number of PGI-positive, vimentin-expressing cells was quantified from five randomly selected high-power fields. The percentage of these cells normalized to total cells (DAPI-stained cells) in the hypercellular areas is shown (D). Data are presented as the mean ± SD. (** p < 0.01) from representative data of 3 independent experiments.

    Article Snippet: Sections were applied with 1:100-diluted rabbit anti-human PGI (#57893, Cell Signaling) and 1:500-diluted mouse anti-human vimentin primary antibodies (#5741, Cell Signaling) at 4 °C overnight followed by washing and incubated with secondary antibodies conjugated with 1:500-diluted goat anti-mouse Alexa Fluor TM 488 (A11001, Invitrogen) and goat anti-rabbit Alexa Fluor TM 568 (A11011, Invitrogen).

    Techniques: Expressing, Immunohistochemical staining, Staining, Incubation